CRISPR Knockout Cell Line Library
abm's CRISPR Knockout Cell Lines are available for any human gene, and arrive ready-to-use for the ultimate convenience. Our expert scientists will take care of the entire CRISPR genome editing process, from sgRNA vector design and delivery, to screening and validation.
Key Features
- Delivered is a cell line with your gene of interest knocked out and supporting QC data.
- Cell lines offered include: 293, 293 T, A549, HeLa, HepG2, MCF7, K562, and U87-MG.
- Saves you time and labor, as we will generate the final knockout cell line for direct use in your experiments.
Can't find your gene of interest in this library? Consider using our Custom CRISPR Knockout Stable Cell Line Generation Service. For knock-in services, we also have a CRISPR Stable Knock-in Cell Line Generation Service.
Popular Products
Additional Resources on CRISPR Technology
Search Knockout Cell Line Library
Knockout Cell Line Library
We offer 7 human knockout cell lines: 293, 293 T, A549, HeLa, HepG2, MCF7, K562, and U87-MG.
Can’t find what you’re looking for? Request a custom cell line with [email protected].
Product Information
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Performance/Data
Click to see our CRISPR Knockout Case Study, including experimental design and supporting data. -
Workflow
The basic workflow for generating CRISPR knockout cell lines. -
Need Help Getting Started?
This handbook outlines guidelines for CRISPR sgRNA design and the experimental procedures needed to achieve a specific gene knockout.
Documents
Top Publications
Regulation of NKT cell-mediated immune responses to tumours and liver inflammation by mitochondrial PGAM5-Drp1 signalling.
Kang, Y J et al.
Nat. Commun. 6:8371 (2015).
DOI: 10.1038/ncomms9371.
Isorhapontigenin (ISO) inhibits invasive bladder cancer (BC) formation in vivo and human BC invasion in vitro by targeting STAT1/FOXO1 Axis.
Jiang, G. et al.
Cancer Prev Res. Published Online First April 14, 2016.
doi: 10.1158/1940-6207.CAPR-15-0338
Clinical significance of SNORA42 as an oncogene and a prognostic biomarker in colorectal cancer.
Okugawa, Y et al.
Gut (2015)
doi:10.1136/gutjnl-2015-309359
